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Sunday Poster Session
Category: Esophagus
Yazan Addasi, MD
Creighton University Medical Center
Omaha, NE, United States
Endoscopic findings | Type of dysphagia | Mucosal disruption post dilation | Linear distribution of diverticula | Associated underlying condition | Hiatal hernia | Biopsy | |
Case A | Ringed esophagus and longitudinal furrows, in entire esophagus, along with multiple small mouth diverticula in upper and middle third of esophagus | Solid food | Significant mucosal tear without perforation | Linear | Eosinophilic esophagitis | Present | Intracellular edema as well as increased intramucosal eosinophils ( >20 eosinophil/hpf) |
Case B | Ringed esophagus and longitudinal furrows, along with narrow mouth diverticula noted in linear fashion in between the rings in the upper esophagus | Solid food | Significant mucosal tear without perforation | Linear | Eosinophilic esophagitis | Absent | squamous mucosa with marked spongiosis and increased eosinophilic infiltrate (30-35 eosinophil/hpf). Superficial clustering of eosinophil as well as eosinophilic microabscesses. |
Case C | Narrow caliber esophagus with rings, benign intrinsic stenosis in upper esophagus, along with multiple small mouthed diverticula noted in upper and middle third of the esophagus | Solid food | Significant mucosal tear without perforation | Uncertain | Gastroesophageal reflux disease | Present | Acute esophagitis with rare yeast forms on PAS stain consistent with candida esophagitis. No significant increase in eosinophils. |
Case D | Widely patent Schatzki ring, along with small and large mouthed diverticula in the lower third of the esophagus. Mild esophagitis. | Solid food | No mucosal disruption post dilation | Linear | Gastroesophageal reflux disease | Present | Mild basal hyperplasia with no evidence of eosinophilic inflammation. |